Morgellons Australia Support Mob☻
an emerging disease that urgently needs to be researched.

May
04

we have had some good success with using an anti microbial treatment made for killing off mould and fungi on plants. so far very good. made from orange peel and yucca plus other things I guess. I like this as it is natural and not at all harsh

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May
04

http://lymedisease.org/news/lyme_disease_views/morgellons-conference-summary.html

Austin, TX (PRWEB) May 02, 2012
A multinational panel of medical and scientific professionals met on March 24 and 25, 2012 in Austin, Texas at the 5th annual Morgellons Medical Conference entitled, “Searching for the Uncommon Thread” to discuss the latest findings on Morgellons disease.

May
04

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3257881/

… ‘The multifactorial etiology of digital dermatitis is not well understood, but spirochetes and other coinfecting microorganisms have been implicated in the pathogenesis of this veterinary illness. Morgellons disease is an emerging human dermopathy characterized by the presence of filamentous fibers of undetermined composition, both in lesions and subdermally.’ …

 

Apr
21

Dr Mayne

Firstly, apologies for ignoring this site for so long. Nothing much has changed for us except we are living in a better place; are happy and therefore healthier. Notwithstanding, Peter’s lesions have stayed. Up until recently he had probably 80% of his body covered with lesions.

I still feel the variation in the obvious level of infection between us is his diet – coffee, sugar and more sugar in the form of sugar, biscuits, lollies and ice cream. Of these I may have A biscuit, not a packet and a small cone of ice cream not a huge bowl. No he isn’t overweight 🙂 I don’t take any caffeine and very little refined sugars, plus I drink chai a spiced tea which contains things like cinnamon, cloves … good cleansing spices.

I will do some more searching and see if I can find some good up to date research information but I was pleased to find that we have a leading researcher here in Australia. Finally some acceptance for Lyme’s Disease as an Australian specific disease ie you get it from Australian species ticks.

I hope you are all keeping well and hope to write more often. Maybe we will see some major breakthroughs?!

peace and happiness

 

Jan
07

http://www.carnicom.com/ph6.htm

MORGELLONS :
pH, CONDUCTIVITY, IONS & LIVE ANALYSIS

Jan
07

There are two new videos .. well I just came across them 🙂 about an interview with a person who claims to have been part of creating Morgellons as a bio weapon.
http://www.carnicom.com/morgobs1.htm – I re-read this article which shows the difference between hair and the fibres.

Sep
25

Released 7/21/09 – Randy S. Wymore, Ph.D.

Statement on Morgellons disease: Center for the Investigation of Morgellons Disease, OSU-CHS.

Morgellons is a condition which has an array of unusual symptoms. The sufferer often has skin lesions which heal very slowly and both the lesions and non-lesioned skin are associated with fibers and other material which seems to be shed from the skin. The skin symptoms are usually associated with intense itching as well as burning and stinging sensations. Many with Morgellons experience intense episodes of fatigue along neurological effects that usually fluctuate in intensity in an unpredictable fashion. The neurological effects can include tremors, peripheral neuropathy, difficulty in focusing thoughts and behavioral changes. Since there are no commonly characterized diseases or disorders that match these symptoms, many with Morgellons have been mislabeled as having a purely psychiatric disorder. This misdiagnosis has caused much needless suffering within the Morgellons community and is unnecessary as a careful skin examination can usually determine whether or not a person has Morgellons. A clinical dermatoscope can be utilized to visualize fibers in the skin. In addition, fiber masses under the unbroken, superficial skin have been observed in every Morgellons patient examined by OSU-CHS clinical faculty. Thus far, only individuals claiming to have Morgellons have been observed to have such fiber masses within areas of otherwise normal appearing skin. To date we still do not know the cause of Morgellons or a definitive treatment plan.

At Oklahoma State University Center for Health Sciences (OSU-CHS), our approach has been to: 1) characterize the physical properties of the fibers by microscopic and spectroscopic analyses, 2) culture bacteria and fungi associated with Morgellons samples, and 3) sequence DNA associated with the samples. This last approach is in an effort to identify any unusual microorganisms or parasites that might be present. Large donations are funding analysis of the fibers, which we are hiring commercial materials analysis laboratories to perform, as well as some electron microscopy at the main OSU campus. These large donations will also fund a significant part of a DNA analysis technique known as ‘shotgun sequencing’. The problem with standard DNA amplification and sequencing, which we do in-house, is that it is time consuming and every single sample must be prepared individually. Plus we cannot effectively look for ‘unknowns’; that is to say we can only look for things we are expecting to find. With shotgun sequencing there need not be any pre-bias; the process will generate thousands of fragments of DNA sequence that can then be analyzed. This will include human DNA fragments, from the host, as well as bacteria and fungi we would commonly expect to find. The hope is that in addition to the many species of microorganisms that live in and on us all, an unexpected parasitic or microbial DNA sequence will also emerge. Once we have a clue to follow, it will be possible to look for that microorganism or parasite in other Morgellons sufferers.

As is often the case in science, things take much longer than an educated guess might expect. It had been hoped that preliminary results from the microscopy and spectroscopy analyses would have been done 2-3 months ago. Unexpected difficulty has arisen with the processing, handling and shipping of the fibers. Even the commercial laboratories have been less than successful at helping us work through these problems. Simply put, the fibers have to be ‘preprocessed’ for much of the testing, and even for those tests not needing preprocessing, the labs want either dozens of identical copies of the material, or a single sample that is immobilized for shipping and yet accessible for analysis. These have proven to be unexpectedly challenging tasks. Still, progress is being made. When a suspect cause has been determined this information will be disseminated to physicians, public health officials and the Morgellons community as quickly as possible.

May
30

Personal Health; The real problem with children’s scalp ills.

By Jane E. Brody
Published: Wednesday, November 9, 1994  NOTE THE DATE

WITH school in full swing, lice and ringworm have once again gone to the heads of the class. But while the families of affected children are most distressed by what they regard as embarrassing if not repulsive infestations, dermatologists say the real problems lie in misdiagnosis and inappropriate and sometimes hazardous treatments.

Ringworm Re-Emerges

A half-century ago, ringworm, a fungal infection that most often attacks the scalp, was a common scourge among school-aged children. And while it lapsed into relative obscurity for decades, ringworm is now back in a more insidious form that is harder to recognize and trickier to treat. In its previous incarnation, ringworm mainly affected Caucasian children and could be detected easily because the fungus that caused it fluoresced under ultraviolet light. The current form of the disease most often attacks African-American children and must be diagnosed by taking scrapings of the scalp and culturing the fungus. Although primarily a disease of school-age children, it is becoming increasingly common among adults and infants, according to Dr. Ilona J. Frieden and Dr. Renee Howard, dermatologists at the University of California in San Francisco.

Ringworm is a misnomer, since the new infection does not form a ring and neither resembles nor is caused by a worm. This fungal infection, known medically as tinea capitis, can be present for months or years before subtle but widely distributed hair loss and inflammation become apparent. Children with ringworm in its early stages may complain about itching and flaking of the scalp, which is often mistaken for dandruff and treated futilely with over-the-counter medicated shampoos. As the infection progresses, a closer look should reveal tiny black dots on the scalp at the hairline; the dots are the remnants of hairs that have broken off within the follicle as a result of the infection. “Misdiagnosis and delays in diagnosis have allowed ringworm to spread unchecked in classrooms, day-care centers and within families,” said Dr. Rebat M. Halder, chairman of the department of dermatology at Howard University Hospital in Washington. The infection is easily transmitted through shared combs, brushes, caps, hair ribbons, barrettes and the like, although simply washing such items in hot soapy water is all that is needed to get rid of the fungus, Dr. Halder said. It can also be acquired from contact with telephone receivers and seat backs that have been contaminated by a previous user.

Children from 3 to 9 years old are most likely to pick it up. While it is more common in inner cities, every school-age child — poor or rich — is a potential target. Genetic factors and certain hair-care practices like tight hair braiding and the use of pomades and oily hair dressings are believed to increase the vulnerability of black children.

The fungus at first attacks nonhairy skin; tight braids can leave parts of the scalp exposed and unprotected, and gluey hair dressings can give the fungus a better opportunity to adhere to the scalp and invade. The current culprit is a fungus known as Trichophyton tonsurans, which is estimated to cause 90 percent of cases in this country.

Unlike the ringworm fungus of yore, this one does not respond to topical treatments, because the organism grows within the hair shaft so that substances applied directly to the scalp cannot eradicate it. Rather, T. tonsurans infections must be treated with oral medication, typically an antifungal agent called griseofulvin taken daily for at least six weeks.

Since many adults are symptomless carriers of the fungus, to reduce the chances of reinfection, when one person in a family is found to be infected, all members of the household should be tested and treated if necessary. Epidemics of Head Lice In the last 15 years, outbreaks of head lice have become commonplace in schools and show little respect for education, social class or degree of cleanliness. Every school year, more than six million cases come to light, a significant share of them representing repeated attacks by these tiny, wingless insects that feed on human blood. Head lice are uncommon among black children. Dr. Mervyn Elgart, chairman of dermatology at George Washington University Medical School in Washington, describes head lice as “more of a psychological problem than a real medical problem.”

Parents understandably cringe when their well-cared-for children are invaded by creepy, crawling creatures. But head lice can become a medical problem if the wrong substances are used to treat the condition or if the appropriate substances are overused.

Because remedies are sold over-the-counter as shampoos, creams and lotions, many parents fail to appreciate the fact that they contain potent insecticides that can become highly toxic if misused. Particularly risky are products that contain lindane, which can sometimes cause seizures or convulsions and brain damage, especially in very young children, even when used according to directions. Although the best-known lindane product, Kwell, is no longer being manufactured, generic forms of lindane are still available.

Louse bites can cause severe itching, and those who scratch for relief can sometimes develop secondary infections. Most commonly, it is not the insects themselves but their oval, yellowish-white eggs, or nits, that are noticed by parents. Undisturbed, the female louse lays three to five eggs a day for about a month. The eggs are attached to the base of hairs, often on eyelashes and eyebrows as well as on the head.

They spread easily from person to person, usually through direct head-to-head contact. But contaminated clothing, bedding, grooming items and headphones can spread them as well, for example, when several children’s coats are hung together on the same hook.

Getting rid of lice requires simultaneous treatment of all infected individuals who are in close contact. Although there is considerable controversy about the best and safest products, most dermatologists now recommend a cream rinse called Nix, which contains permethrin, a pesticide minimally toxic to mammals. A one-time treatment kills only the adult lice, but the chemical remains active on the hair and scalp for 10 days or longer (even if you shampoo and swim daily) and can kill adult lice that emerge later from the impervious eggs.

Deborah Altschuler, president of the National Pediculosis Association in Boston, said there were problems with all the available products, with none living up to manufacturers’ claims for effectiveness.

Dr. Elgart said that keeping potentially contaminated clothing and furnishings away from people for about five days spells death to head lice, which can live only as parasites. “You don’t have to boil or dry-clean everything in sight,” he insisted.

http://www.nytimes.com/1994/11/09/us/personal-health-the-real-problem-with-children-s-scalp-ills.html

Apr
21

I still feel there is a strong link between Lyme’s Disease and Morgellons. I have been reading stuff on Lyme’s in Australia. I have been caught up today going from site to site reading about Lyme Disease, ticks and more …

[http://medent.usyd.edu.au/fact/lyme%20disease.htm#conclu]

Conclusions – Australia

There are some major differences between Australia and the endemic areas of the northern hemisphere with respect to the natural history of LD:

No ticks of the I. persulcatus complex, the principal vectors to humans in the northern hemisphere, occur in Australia. In eastern Australia, the logical candidate vector would be I. holocyclus which has a wide host range and is the most common tick biting humans. It was unable to transmit a North American strain of B.burgdorferi but an association with a so far undiscovered Australian spirochaete can not be excluded.

None of the mammal species identified as reservoir hosts in the northern hemisphere are present in Australia. There are reports of spirochaetes in Australian native animals, and a local mammal could be a reservoir host for an indigenous spirochaete that occasionally infects humans through a tick vector and produces a clinical syndrome similar to LD; however, no spirochaete was detected in the 12,000 ticks or animals processed.


Summary

The diagnosis of LD outside known endemic areas cannot be based solely on serological tests especially if they fail to conform with internationally accepted criteria, because of the high incidence of false positive results.

A clinical diagnosis in a non-endemic disease area (especially of Stage II or III disease), is difficult to support without isolation of the causative agent from the patient, from other patients with similar illness or from a known vector in the region.

The existence of LD in Australia will remain controversial until an organism is isolated from a local patient and fully characterised, or until a tick-borne organism can be shown to be responsible for the human infection. If it exists it shares few of the epidemiological or clinical characteristics of US or European patterns of LD.’

Ability to transmit diseases (vector competence) [from http://www.tickalert.org.au/ixholdet.htm]

In general terms a vector may simply mechanically transport a pathogen without it replicating, or it may both transmit and replicate a pathogen. Many viruses, bacteria, rickettsia and protozoal organisms are capable of replicating within ticks. This is known as “biological” transmission and this mode of transmission is the one that applies most commonly to ticks.

For hard (Ixodid) ticks it is virtually impossible for mechanical transmission to occur on its own (i.e. without some replication), as they tend to not take multiple blood meals from different hosts in the one stage. Passing an organism between tick stages (trans-stadial transmission) seemingly requires that an organism can also replicate within the host.

In the case of Ixodes holocyclus there is much speculation about transmission of various pathogens, with the exception of the spotted fevers which are now well recognised. Saying that an arthropod is a competent vector (generally) requires several findings:

(1) frequent isolation of the suspected pathogen;

(2) showing that the distribution of the suspected vector overlaps with a diseased population (be it human or animal);

(3) showing that host-seeking and blood-feeding coincide with the disease incidence;

(4) showing that disease incidence varies with host choice; and

(5) showing the capacity for vector competence in the laboratory.

Bacterial DiseasesRickettsiae

These days there is not much disputing that I. holocyclus is the main vector for R. australis (Rickettsial Spotted Fever). Unfortunately vector competence work is still required to confirm its status. (S. Doggett pers. com.).

Despite the lack of vector competence studies, the paralysis tick (I. holocyclus) is commonly mentioned as a potential vector of Q-fever (caused by the rickettsial organism Coxiella burnetii). The ornate kangaroo/wallaby ticks Amblyomma triguttatum s.l. have also been implicated in Q-fever (Russell and Doggett, 1995).

Spirochaetes

Some vector competence studies have been undertaken on I. holocyclus with respect to the Lyme disease pathogen Borrelia burgdorferi sensu stricto (with a United States strain). These suggested that the tick can not transmit this strain of spirochaete (Piesman and Stone, 1991).

Despite this, there is a strong belief that some kind of Lyme-like spirochaete causes a Lyme-like disease in Australia and that it is carried by the most common tick afflicting humans, Ixodes holocyclus. At the Royal North Shore Hospital in Sydney the Tick-borne Diseases Research Unit continues its study into this issue.

Cases of Lyme-like disease are being diagnosed on the basis of clinical signs (often musculoskeletal, chronic fatigue, neurological and dermatological), exclusion of other infections, serology (which is supportive but not conclusive), and response to antibiotic treatment (initially, antibiotics may cause a worsening of symptoms [the Herxheimer reaction], presumably as spirochaetes are destroyed, and this in part supports the diagnosis).

The Australian form of Lyme-like borreliosis is associated with a different clinical picture from true Lyme disease of North America. See also the Westmead Hospital Dept of Medical Entomology site for their interpretation on the status of an Australian Lyme-like Disease.

For comparison with North American Lyme borreliosis go to healthlinks.washington.edu.

Viral Diseases

So far, no viruses have been isolated from I. holocyclus (Russell and Doggett, 1995). This does not however exclude the possibility that such diseases may be found in the future.

Protozoal Diseases

So far no protozoa have been isolated from I. holocyclus. This does not however exclude the possibility that such organisms may be found in the future.

Apr
17

Morgellons mystery

Left: A Morgellons fiber: 0.1 mm. Right: Morgellons sufferer Cindy Casey shows the scars left by the disease on her arms. The rest of her body bears similar marks. -Photo by Will Atkins

Right: Morgellons sufferer Cindy Casey shows the scars left by the disease on her arms. The rest of her body bears similar marks. -Photo by Will Atkins Corinne Carson 15.APR.09

Conference on strange disease held in Oak Hill area It sounds like science fiction. People report strange unknown fibers emerging from under their skin in hues of blue, red, white, black, even turquoise. Researchers report that the fibers are made of cellulose, which is not normally found in the human body, and some have even found fibers coated in metal. Some fibers have been observed to move. The fibers are accompanied by an intolerable feeling of something crawling, stinging or biting under the skin, a feeling that gets worse at night. This torment is accompanied by a variety of other symptoms, including skin lesions and “brain fog” and the symptoms are sometimes shared by whole families, and even their pets.

The condition, known as Morgellons Disease, is not officially recognized by the mainstream medical community and sufferers are often diagnosed with other skin illnesses or told that they are suffering from a psychological condition known as delusions of parasitosis � in other words, that it is all in their head.

Although the mainstream medical community does not recognize the illness, on one day out of the year, the often self-diagnosed Morgellons patients and sympathetic medical professionals and allies gather for their international conference. This year, that conference was held April 4th in Southwest Austin at the Westoak Woods Baptist Church.

The conference is sponsored by the Charles E. Holman Foundation, a non-profit that states its mission is to support “research, education, diagnosis and treatment of Morgellons Disease” and included lectures from sympathetic medical professionals and researchers. Charles Holman is the late husband of Cindy Casey RN, a Morgellons patient. He founded the “New Morgellons Order” to raise awareness of the disease after accompanying his wife to a number of doctors’ appointments and watching as she was told she was crazy. The foundation was renamed in his honor when he died of a heart attack in 2007 and his wife Casey took over as the executive director.

Cindy Casey’s story is like many others. She began experience symptoms of chronic fatigue and occasional skin lesions as far back as 1997. By 2003, the lesions had become so prevalent and excruciating, with the characteristic fibers emerging from her skin and the intense itching, pain and cognitive problems or “brain fog” that she decided to seek treatment.

Like many “Morgies” as Morgellons patients sometimes call themselves, Casey was told by several dermatologists that she suffered from delusional parasitosis, a condition in which the patient hallucinates the sensation of bugs crawling under the skin, often accompanied by skin wounds caused from excessive itching or picking at the skin.

In an intermission during the conference, Casey rolled back her sleeve and lifted her pant leg to reveal arms and legs covered with numerous lesions and discolored whiter skin where she said previous lesions had healed. “They start out as little pimple marks and then they become open wounds, so I have them in all different stages,” she said.

Other patients came up, lifting their skirts and pant legs to reveal the same lesions dotting their skin. Some had lesions under white cotton gloves or hidden under handkerchiefs tied over their head.

Of the lesions, Casey said, “They are so painful it’s hard to describe � the itching is worse. It’s just this insatiable itch that you can’t even describe. I’ve had poison ivy and those things itch bad but this is just, it’s just so bad it makes you want to come unglued.”

Casey, like many others at the conference, credits Ginger Savely, DNP, a nurse practitioner famous for treating the controversial Morgellons and Chronic Lyme diseases, for finally showing her respect and compassion, and for giving her treatment that she says has reduced the number of lesions and the severity of her pain and itching.

Savely, who has a PHD in nursing, is practically worshipped in Morgellons and Lyme circles. She gave two lectures at the conference, one in which she described case studies of various Morgellons patients she has treated, and another discussing treatment options for the disease. While acute Lyme disease has finally become recognized by the medical community, Chronic Lyme, in which the Lyme symptoms persists after the normal course of antibiotics, is still controversial. Many doctors do not believe it exists.

Not only is Chronic Lyme real, Savely maintains, but it has a strong association with Morgellons. She discovered the connection while treating Chronic Lyme patients in her practice in Austin. She found that approximately 10 percent of her chronic Lyme patients had the fibers, lesions and skin-crawling sensations characteristic of Morgellons. As news of the Morgellons phenomenon spread over the Internet, she started hearing from more patients who came to her for Morgellons treatment. She says overall, about 95 percent of Morgellons patients she has treated are co-infected with Lyme disease.

Savely postulates that Lyme may weaken the immune system, making patients more vulnerable to whatever pathogen causes Morgellons, but she admits she does not know what that pathogen is.

This is what really upsets many in the mainstream medical community. They criticize her for prescribing heavy doses of a variety of medications for extended periods, outside the normal medical guidelines, when she doesn’t even know what she is attacking or which of her treatments will work.

“I’ve been told that I’m just randomly giving these different treatments to the patients without knowing what the pathogen is and that’s really irresponsible. But I think what’s irresponsible is ignoring suffering people and not treating them at all,” Savely said, and the crowd erupted into applause … and treat she does. She first works to heal any co-infections such as Lyme disease, and then prescribes simultaneous heavy doses of antibiotics, de-wormers, anti-fungals and various topical creams, trying various medications on a patient until she finds the ones that work, sometimes treating patients with these medications for years at a time. “Every patient is unique. The more I treat this illness the stupider I get,” she said, explaining how she will often find a treatment that works well for a time and then just stops working, and that what works well for one patient, won’t necessarily work for another. Savely doesn’t know why the de-wormers are effective, since there are no “worms” found in Morgellons patients but suspects that they are in someway toxic to the pathogen. When she prescribes them, she says patients experience an immediate worsening of lesions and of the crawling, biting and stinging sensations, which she attributes to a “mass-exodus” of the fibers out of the skin. “I do warn patients, you’re going to get worse before you get better. That’s to be expected, and the majority do get worse before they get better.” But she says most patients do get better, although not all become completely symptom free. One of Savely’s success stories stood up, a woman who Savely said was at one time totally debilitated by the condition but has now been symptom-free for three years.

Despite the success stories, some in the medical community feel Savely’s treatments are dangerous. She was forced to leave the state of Texas when the medical board put pressure on her sponsoring physician and she could find no other Austin physician to work with. She now practices in San Francisco, California where the laws governing medical treatment are more liberal. She says she carefully monitors her patients’ liver function and blood count over the course of treatment and has seen only on very rare occasions, very minor adverse affects such as a small elevation in liver function, which she says she easily reverses by reducing the medication. “It sounds scary to the pharmacist and they will try to scare you, but I just haven’t seen the problems, and people tolerate them (medications) very well,” she said.

Although Savely is willing to treat the disease now, everyone agrees that finding the cause of the disease is necessary to find the most effective treatment. The Center for Disease Control (CDC) is currently investigating Morgellons but they have not published any findings. But other researchers are also looking into the cause. One such researcher who spoke at the conference is Dr. Raphael Stricker, MD, a clinician who, like Savely, treats Chronic Lyme and Morgellons patients in California.

Morgellons skeptics dismiss Stricker’s research because he was fired from the University of California in 1990 for allegedly suppressing data in a 1985 research paper on AIDS that would have contradicted his hypothesis. But Stricker has consistently denied the charges and Lyme and Morgellons message boards abound with patients posting messages in his defense. Stricker presented research he conducted along with Savely and Vitaly Citovsky, Ph.D of the State University of New York at Stony Brook, in which they tested lesions from seven Morgellons patients as well as biopsied skin samples from a healthy control population.

They found agrobacterium in all seven Morgellons patients and none was found in the control group. Agrobacterium is a bacteria that infects plants, causing Crown Gall disease in trees, and it is the only known organism to engage in lateral gene transfer among kingdoms, transferring genes from the plant kingdom to the animal kingdom. The bacterium produces copious amounts of cellulose fibers, and Stricker and others have found the fibers found in Morgellons patients to also be made of cellulose, a plant material.

Skeptics point out that cotton is also made of cellulose fibers. But Forensic scientist Ron Pogue of the Tulsa Police Crime Lab in Oklahoma told ABC News that he checked a sample of Morgellons against every known fiber in the FBI database and found no match.

The lab’s director, Mark Boese, told ABC that the fibers are “consistent with something that the body may be producing.” He added, “These fibers cannot be manmade and do not come from a plant. This could be a byproduct of a biological organism.”

Stricker has a theory, but acknowledges a lot of research needs to be conducted to see if his theory is correct. He speculates that agrobacterium, which is found in the soil and also in a high percentage of ticks, is the cause of Morgellons, and that the reason many are co-infected with Lyme is that they may contract Morgellons from the same tick that gave them the Lyme disease. Furthermore, he notes a very curious finding. Telomerase is an enzyme that ads more DNA to the end of a chromosome after cell division. “Every cell and species has telomerase,” said Stricker. “There have been comparative studies of telomerase in different organisms and it’s been shown that the telomerase in Lyme and agrobacterium are exactly the same.” He continued, “This is a peculiar coincidence because telomerases are usually completely different among different species and types of bacteria. This suggests when there is co-infection with Borrelia, which causes Lyme disease, and with agrobacterium, the two bacteria can actually help each other by adding pieces of DNA to each other’s chromosomes and this could cause variation in both bacteria, that could then lead to changes in what the bacteria are doing in the human body.

It’s a very interesting possibility that also needs to be examined in further detail.” He added, “My speculation, and it still is speculation, is that Morgellons Disease may represent the first known human illness caused by a plant bacterium. And in fact, when people say they have these fibers attached to the skin that don’t come out, that are very adherent, in a way they are kind of turning into plants.” He also noted that his research has found some fibers to have a metallic coating, which could explain why many people have witnessed these fibers moving. He said the metal could become charged with electricity, causing the fibers to move.

“The movement is real,” said Casey. “Most everyone you can talk to has seen these things move. It’s almost alien. It’s like your worst science fiction movie. A lot of us will get together and you can put them on the end of a tweezers and see them moving around almost, like a cobra dance.” But getting together, even if it is just to commiserate about the movement in the fibers, is something that seems to help these patients.

Savely said, that in addition to light exercise, nutrition, and her treatment regimen, she urges her patients to seek out human contact. Because some are afraid Morgellons could be contagious, (there is no evidence to show it is contagious under casual contact) and other are ashamed of their disfiguring lesions, like modern day lepers, many sufferers isolate themselves. “I think human touch is so important, said Savely. “You can’t go with out it. I’ve had patients come up to me and I give them a hug and they start to cry and they say, ‘That’s the first time I’ve touched a human being in five years.’ How can you live like that? You know you can’t. You can’t isolate yourself. You need to be around your family now more than ever.”

Look for a follow up on Morgellons in Austin in a future issue of the Gazette.

– Oak Hill Gazette